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In contrast to the oxidation of reduced roGFP2, AtGrxS15 did not catalyse the reduction of oxidized roGFP2 in the presence of GSH (Supplementary Fig

Clarifying the spectrum of driver oncogene mutations in biomarker-verified squamous carcinoma of lung: lack of EGFR/KRAS and presence of PIK3CA/AKT1 mutations

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It achieves this by precisely activating ghrelin (GHS-R1a) receptor pathways without significantly elevating other pituitary hormones, such as cortisol or prolactin, in experimental models