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Hyperprolactinemia interferes with the activity of gonadotropin-releasing hormone (GnRH) neurons and is a recognized cause of functional hypogonadism ( At the central level, excessive prolactin inhibits the synthesis and pulsatile secretion of hypothalamic gonadotropin-releasing hormone (GnRH), thereby reducing the release of pituitary gonadotropins, luteinizing hormone and follicle-stimulating hormone, ultimately leading to hypogonadism ( In women, prolactin has a significant inhibitory effect on ovarian steroid production, leading to reduced synthesis and secretion of estradiol and progesterone ( In men, hyperprolactinemia is accompanied by hypogonadism and androgen deficiency, which can lead to unfavorable changes in the body composition

95% UI, 7,718.89 to 146,211.01)

Li Y, Li C, Luo T, Yue T, Xiao W, Yang L, et al

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- Basic research and clinical perspectives-

Researchers employ Cagrilintide in various in vivo animal models, including diet-induced obese (DIO) rodents and genetic models of metabolic dysfunction, to characterize its effects on key physiological endpoints