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Introduction Tobacco smoking causes morbidity leading to disabilities and reduced life quality, and causes 12% of all deaths globally.1 Self-reported smoking information is subject to recall bias and understatement by the patient.2 ,3 Furthermore, it does not capture subtle, but important variation in smoking behaviour due to variation of number of inhalations per cigarette, depth of inhalations, respiratory retention of the smoke and heaviness of second-hand on top of first-hand smoking.4 ,5 Therefore, self-reported cross-sectional smoking information produces underestimated risk estimates, as suggested in genetic studies using a smoking-related genotype of nicotinic acetylcholine receptor ( CHRNA3 ) as proxy for the lifetime exposure to tobacco smoke6

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