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Through the action of mitotic kinases such as Mps1 and Cdk1, key checkpoint proteins including Bub1, BubR1, Mad1, Mad2, and Cdc20 are sequentially recruited to unattached outer kinetochores, primarily via phosphorylation-dependent interactions 2

Non-invasive molecularly-specific millimeter-resolution manipulation of brain circuits by ultrasound-mediated aggregation and uncaging of drug carriers

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GWAS analysis helped distinguish the genetic polymorphisms that modulate either the steatosis, fibrosis, or both in NAFLD patients

Shifman S, Bronstein M, Sternfeld M, Pisante-Shalom A, Lev-Lehman E, Weizman A, et al