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UpToDate (updated as of 2025).

This suggests that a resistance exercise-related mechanism of inducing plasma NO, perhaps due to increased shear stress that triggered an up-regulation in NO-cGMP signaling along with a slow, sustained release of NO from GSH + CIT, may be a conceivable candidate for this response

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rather, the WHO recommends integrating salt/sodium reduction strategies with parallel salt iodization programs [46]

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