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Although oxidative stress is known to induce post-translational modifications/damage to mitochondrial proteins [25, 26], it is currently unknown whether mitochondrial structure and function in airway epithelial cells are affected by cigarette smoke [27, 28], and whether these changes are present in COPD epithelium

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the present invention provides a filter for tobacco smoke inhaling/generating/producing device, the said filter comprising three sections placed longitudinally one after another wherein, the first section comprising cellulose acetate fibre acting as a mouth piece, the second section comprising activated charcoal selected from group consisting of charcoal particles having grain size ranging between 25 mesh and 100 mesh for effectively reducing p-benzosemiquinone, a highly reactive major harmful oxidant from the mainstream of cigarette smoke and the third section comprising cellulose acetate fibre located closer to the tobacco portion of the cigarette also acting as a barrier between the activated charcoal and tobacco length of the first section is in the range of 10 to 14 mm length of the second section 4.5 mm to 35 mm which is dependent on the grain size and/or amount of charcoal used and length of the third section is in the range of 2to 3 mm