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Taken together, these findings underscore the critical role of SIRT1 in modulating inflammation-driven ferroptosis through NF-B, NLRP3 and AMPK signaling

Upon binding of the ligand, PPARs heterodimerize with retinoid X receptors and bind to cis-acting DNA elements (PPREs) to increase gene transcription

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Results: DSIP-CBBBP demonstrates a capacity to modulate neurotransmitter levels, indicated by changes in 5-HT, glutamate, DA, and melatonin

FOXO4-DRI has been suggested to prevent normal FOXO4 binding to p53, eliminate senescent cells, enhance organ function, and reduce cell death progression

Experimental results indicate reduced oxidative damage and improved mitochondrial function