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Activate a cancer-like program to survive independently of efficient blood circulation 57,58

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Consequently, this delays wound healing (Zhao et al., 2020), inhibits the phagocytic function of macrophages, and impedes macrophage polarization from the pro-inflammatory M1 to the anti-inflammatory M2 phenotype

In corneal epithelial cells, this allows quick response to migration signals during wound healing

GA also stimulates the expressions of TJ proteins, ameliorates cell apoptosis, and oxidative stress, and suppresses the activation of the NF-B/MAPK pathway to alleviate LPS-induced intestinal inflammation in Caco-2 cells [345]

Tamoxifen Activates Nrf2-dependent SQSTM1 Transcription to Promote Endometrial Hyperplasia