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As noted, depression is strongly connected to chronic inflammation, and NSAIDs, by inhibiting cyclooxygenase (COX) activity and reducing prostaglandin (PG) synthesis, indirectly regulate the NF-B signaling pathway, thus decreasing the release of pro-inflammatory cytokines like interleukins and TNF-, which may mitigate the inflammatory responses involved in depression (140)

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However, more importantly, in the current study, the correlation between MG presence and enhanced cytokine output with lower neural cell death is suggestive of their neurotrophic role in the ischaemic retina

Leptin-activated hypothalamic proopiomelanocortin (POMC) and agouti-related protein (AgRP) neurons located in the arcuate nucleus are typical (126, 127)

Critically, the excessive buildup of iron within the brain establishes a self-sustaining cycle: iron accumulation promotes -amyloid (A) aggregation and tau hyperphosphorylation (Tran et al., 2022), while A plaques in turn sequester iron via their metal-binding domains, increasing local Fenton reaction activity (Tang et al., 2024)