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2014), affecting both the balance of hepatic free fatty acids and the expression of key lipid metabolism genes in the liver (Suciu et al

While mTORC2 primarily promotes cell proliferation and survival through the phosphorylation of AKT, many tumors need mTORC1 to maintain their ability to control protein synthesis and cell growth, as well as to coordinate nutrient and energy supply to ensure that they undergo unrestricted cell division (103)

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Fibroblast Recruitment and Proliferation: Fibroblast migration toward the injury site increases in BPC-157-treated models, producing earlier and more abundant collagen matrix deposition

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