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Ohara T

These alterations in levels have likely taken years to manifest resulting in well-established functional deficits

For example, S1PR1 can utilize Rho kinase and PI3K-Akt1 signaling to modulate peritoneal macrophage migration, whereas S1PR2 can stimulate cAMP production by activating NLRP3 inflammasomes but inhibiting AKT phosphorylation, and thus inhibit macrophage migration [141]