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10.1016/S0140-6736(71)90970-6

Excess iron has long been postulated as a risk factor for developing liver fibrosis and cirrhosis, and recently direct evidence has emerged with respect to the pathogenic role of ferroptosis in iron overload-induced liver damage and fibrosis [14]

R2*, which is additively sensitive to a wider range of causes of tissue changes, including cell-count, shows more consistent longitudinal increases during PD than QSM 176,177 , as does free-water imaging of the SN 178

Systemic lupus erythematosus (SLE) therapy: the old and the New

Association of clinical complete response (cCR) after preoperative chemoradiation and pathological complete response (pathCR) in patients with gastroesophageal cancer (GEC) and indispensability of trimodality therapy (TMT)

The superfamily of GPCRs is one of the largest families of proteins in the mammalian genome and shares a conserved structure composed of seven transmembrane helices (Fredriksson and Schioth, 2005)