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From a pathophysiological perspective, dietary fructose promotes DNL, impairs fatty acid oxidation, and triggers hepatic inflammation, thereby clearly fueling hepatic insulin resistance (reviewed in [99])

The mechanisms underlying AF in patients with metabolic disorders are aetiology-dependent, emphasizing the need for tailored therapeutic strategies to treat the arrhythmia

Alternate day fasting improves physiological and molecular markers of aging in healthy, non-obese humans

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the bronchodilator ipratropium and the calcium channel blocker verapamil) have been proposed as substrates 19,20 , though these compounds, alongside a number of other cationic drugs, were shown to be poor substrates for OCTN2 21