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It can induce effective cell death and stimulate immune responses in apoptosis-resistant GC cells, particularly by promoting the activation of dendritic cells (DCs) and cytotoxic cluster of differentiation 8-positive T cells (CD8 + T cells) through the release of damage-associated molecular patterns (DAMPs)

So why is this a good thing

Glycine, usually protective, may paradoxically increase oxidative stress

It happens when the muscle spasms and becomes tight, disrupting the flexibility of the mechanical interface

Conversely, free radicals can increase the concentration of glutamate in the synaptic cleft by the release of glutamate or blockade of its re-uptake

d: Graphic representation of similarity for cobamamide (left) and RDV (right) generated by RDkit