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We observed that pmk-1(km25) and sek-1(km4) mutants fail to suppress SKN-1 activation and instead exhibit increased activation with amdh-1 knockdown (fig 2a,b)

doi: 10.1111/ppl.12292 110 LouisN.DhankherO

& Gray, S

All of these factors combine to make the CNS particularly vulnerable to oxidative stress, which can lead to cell dysfunction and even death, and is closely associated with the development of multiple CNS diseases