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doi:10.1186/s12970-015-0067-x Bellanti F, Coda ARD, Trecca MI, Lo Buglio A, Serviddio G, Vendemiale G

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71 Interestingly, metabolite profiling of cells treated with Cu ionophore presents a time-dependent increase in the dysregulation of many TCA cycle-associated metabolites, and inhibiting the electron transport chain complexes I and II significantly reduced Cu-induced cell death

Following endogenous growth hormone release, insulin-like growth factor-1 (IGF-1) mediates many downstream physiological processes involved in normal growth hormone biology

In addition, lysosomes also participate in this crosstalk by regulating autophagy and mitophagy, processes essential for clearing damaged organelles and maintaining cellular homeostasis (49, 50)

A secretagogue nudges an upstream node and lets physiology set the resulting IGF-1 level within feedback limits