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Hence, when an antioxidant can significantly reduce the level of ROS (most especially mitochondria ROS), mitigate oxidative damage, and improve mitochondrial bioenergetics of FRDA mouse models, it justifies further research in this field to comprehensively assess its therapeutic potentials in FRDA

It should also be noted that most of the animal models used (other than Gulo knockout mice and guinea pigs) can also synthesize their own vitamin C

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Numerous hydrophobic interactions, hydrogen bonds and salt bridges stabilise GSH binding (Fig

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