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DAlessio FR, Craig JM, Singer BD, et al

Raitan DJ, Talbot JM, Fisher KD (eds.)

In particular, they have used complementary models of CFTR silencing and blockade (by the CFTR inhibitor, CFTRinh-172) in freshly isolated human umbilical vein endothelial cells (HUVECs)

PLoS One, 13 (8), e0202962

Pulmonary ARDS is usually characterized by extensive ground-glass opacification and asymmetric lung consolidation, while extrapulmonary ARDS is characterized by symmetrical ground-glass opacification distributed in perihilar regions ( 2.3 Heterogeneity in the onset time of ARDS The Berlin definition sets the onset of ARDS as within one week of the occurrence or exacerbation of the underlying injury ( 2.4 Biomarker heterogeneity Biomarkers of ARDS can not only indicate the pathogenesis and progression of the disease, but also conduce to identify ARDS subgroups and provide targeted treatment ( In ARDS, RAGE reflects alveolar epithelial damage, whereas Ang-2 is a marker of endothelial cell activation and increased capillary permeability

They are interdependent