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Epidemiological correlates of diabetic neuropathy

Conversely, individuals without family history may develop cognitive decline due to cumulative exposomic influences (Monaco et al., 2025)

It contains an extended N-terminal structure of 13 amino acids and a replacement of the glutamic acid at residue 3

Amantadine as treatment for dyskinesias and motor fluctuations in Parkinsons disease

The untargeted metabolomics experiments revealed that GTN affects several important metabolic pathways involved in maintaining redox homeostasis, amino acid metabolism, and purine metabolism ( Figure 3 )