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(82) developed a mouse MI model and, through transcriptomics and network analysis, uncovered alterations in metabolic pathways during MI, providing new perspectives for tissue-specific therapeutic targets

Therefore, collectively, these findings suggest that administering NAD + precursors prevents oxidative stress and mitochondrial dysfunction, and hence may be a useful therapeutic target for ALS

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Leuk Res 70:2024 Iovino L et al (2020) Thymic reconstitution after damage is influenced by zinc status

While this can be beneficial in targeting cancer cells, excessive autophagy in normal cells can lead to the degradation of essential cellular components and cell death

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