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Reactive oxygen species (ROS) may directly alter vascular function or cause changes in vascular tone by several mechanisms including altered nitric oxide (NO) bioavailability or signaling

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For instance, depletion of mGSH results in Trx2 oxidation (Zhang et al., 2007), while hypercholesterolemic pigs with selective depletion of mGSH in heart mitochondria, exhibit decreased levels of the mitochondria-specific antioxidant enzymes such as SOD2, Trx2, and Prx3 (McCommis et al., 2011)

GR activity was determined with 100 M GSSG [19]

In lung cancer cells, GPX3 acts as an inhibitor of proliferation, migration and invasion of tumor cells by suppressing ROS-mediated NF-B signaling (27,39)

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