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In the context of pSS progression, SG destruction is common due to the chronic inflammatory environment and ultimately leads to the formation of fibrotic matrix
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It can be seen that tanshinone IIA can regulate the number of fibroblasts in tendon cells and collagen in tissues, and also regulate the balance of oxidation and antioxidants in the damaged area, reduce the damage of oxidative stress to the extracellular matrix, and protect chondrocytes
[84] demonstrated that in colorectal cancer, suppression of the MondoATXNIP axis promotes glucose uptake and glycolysis, leading to the induction of highly glycolytic, Th17-like Tregs
