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Normally, FOXO4 prevents p53 from triggering apoptosis in aging cells, allowing them to persist and contribute to age-related diseases (1)

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Since the discovery of these channels, experimental studies seek the structural detail of each subfamily to reach a greater understanding of the modulatory mechanisms, as well as the action of TRP antagonists and agonists in the respective binding sites or through indirect stimulation, to broaden the understanding of the roles of this superfamily in a physiological and pathological context