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A commonly agreed upon the fact in the pathogenesis of SLE is the dysfunction of T cells, B cells, dendritic cells, macrophages, and neutrophils, likely triggered by genes and/or the environment [4]

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Short hairpin RNA targeting Twist1 suppresses cell proliferation and improves chemosensitivity to cisplatin in HeLa human cervical cancer cells

In general, our results showed that ethanol exposure may reduce the antioxidant capacity in the hippocampal of WT mice, leading to the occurrence of neuroinflammation, while in APP/PS1 mice, ethanol exposure further worsened this process by exacerbating the pathological progression of AD