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The efflux of cytochrome c from the mitochondria into the cytoplasm triggers the activation of pro-caspase-9, which in turn activates pro-caspase-3, ultimately leading to apoptosis [85]

Despite being a surgical intervention, DBS highlights the broader promise of MF-based technologies in the field of neuromodulation, offering insights into non-invasive alternatives for targeted brain stimulation

Endoplasmic reticulum-bound ribosomes reside in stable association with the translocon following termination of protein synthesis

Glutamate excitotoxicity, oxidative stress, and neuroinflammation are signaling pathways that might cause an increase and/or decrease of proapoptotic and antiapoptotic proteins, respectively

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If the fusion partner contains the same affinity tag as the protease, this simplifies purification of the target protein