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Of particular interest was another clinical observation that a WD patient with the G54S PRNP variant presented with very severe symptoms, while his older sister, who carried the same ATP7B genotype but was clinically asymptomatic, lacked this variant allele 22

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But both rest on a foundation of pivotal, replicated human trials that GHK-Cu simply does not have for this indication

Researchers continue examining interactions with: .Cytokine signaling .Cellular stress responses .Immune-cell communication .Oxidative stress pathways .Tissue remodeling mechanisms These studies aim to understand how cells regulate inflammation during repair processes, rather than establish clinical effects