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PMC 3995271

There are other options, too, like intensive outpatient programs and partial hospitalization programs

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doi:10.1111/resp.12611 British Lung Foundation

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These age-related alterations are abnormally increased in COPD patients owing to several reasons, including telomere shortening, cellular senescence, activation of the phosphatidylinositol-3-kinase (PI3K)/mammalian target of rapamycin (mTOR) signaling pathway, defective DNA repair, abnormal microRNA patterns, epigenetic alterations, decreased anti-aging molecules, mitochondrial dysfunction, impaired autophagy, immunosenescence, and stem cell exhaustion (59,60)