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Funding This work was supported, in part, by grants from the National Institute of General Medical Sciences (R35GM147101) to P.M., and the Breast Cancer Research Foundation of Alabama to P.M

An important challenge for future studies will be to define which nodes within this multilayered GSH-centered defense network represent true therapeutic vulnerabilities across different tissue and tumor contexts

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Studies in cultured rat hepatocytes and perfused livers

For example, whatever the radiation quality, an increase in the endogenous GSH pool may inhibit sister chromatid exchange [22], increase exchange aberrations [19], or reduce the frequency of aberrant metaphases [18]

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