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They mitigate the oxidative damage to membrane phospholipids, such as phosphatidylcholine (PC) and sphingomyelin (SM), and restore ceramide (CER) synthesis, which promotes apoptotic pathways in melanoma cells without harming healthy melanocytes 162

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At the same time, Caspase-1 processes Gasdermin D (GSDMD), thereby allowing the N-terminal GSDMD domain to form pores in the plasma membrane, which contribute to the egress of IL-1 and also cause the lytic form of cell death known as pyroptosis 10,11,12,13