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[21] Furthermore, an animal study in ewes demonstrated that a B 12 , folate and methionine restricted diet around conception, resulted in offspring with higher adiposity, blood pressure and insulin resistance which could be accounted for by altered DNA methylation patterns

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From these data we can reasoned that by selectively targeting GSH content of cancer cells we can induce apoptosis either directly or by combining therapies with redox antineoplastic agents

Thus, prolonged postnatal vB12 treatment was able to rescue the PPI deficit in Tbx1 +/ mice

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