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doi: 10.1016/j.pnpbp.2023.110740 175 HalsteadSSiskindDAmftMWagnerEYakimovVShih-Jung LiuZet al

the NR1H4 variants cause loss of BSEP expression, leading to the accumulation of toxic bile and hepatocellular damage, with rapidly progressive intralobular cholestasis in the neonatal period [1, 42, 48, 49]

doi: 10.1016/j.rdc.2022.03.005 7 BakerJFGhioAJ

Liang and colleagues demonstrated that mouse aortic endothelial cells from animals fed a high-Hcy diet exhibit significant ENaC activation, accompanied by impaired endothelium-dependent relaxation (EDR) [85]