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These results are further supported by previous studies dissecting the mechanism of TLR-mediated apoptosis through the RIP1 signaling pathway, where authors have shown that TLRs can activate the RIP/FADD/caspase-8-axis through their common adaptor protein TRIF to ultimately promote cell death ( Our study is set in a TME that is heavily inflamed due to neutrophilic influx via robust COPD-mediated TLR activation