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However, in a small percentage of adult patients it may initiate chronic atrophic inflammatory changes of the corpus or antrum of the stomach accompanied by the increase in gastrin expression and release, enhancement of cyclooxygenase-2 (COX-2) expression and prostaglandin (PG) generation as well as by numerous morphological and biochemical changes leading to the transformation of mucosal cells into malignant cells

Iron brain menace: the involvement of ferroptosis in Parkinson disease

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Glutathione depletion resulting in selective mitochondrial complex I inhibition in dopaminergic cells is via an NO-mediated pathway not involving peroxynitrite: implications for Parkinsons disease

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mTORC1 as a regulator of mitochondrial functions and a therapeutic target in cancer