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Ketamine-induced neurotoxicity blocked by N-Methyl-d-aspartate is mediated through activation of PKC/ERK pathway in developing hippocampal neurons

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The selectivity of -adrenoceptor antagonists at the human 1, 2 and 3 adrenoceptors

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Although stress and inflammation increase KYN availability via the activation of IDO and TDO, downstream flux through the KP is critically determined by the activity of KAT and KMO