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Cancer is a complex, multifactorial disease whose risk of development and progression depends on both intrinsic (oncogenic signaling, epigenetic alterations, genomic instability, and disturbed cell metabolism) and extrinsic (tissue damage, infections, and mutagenic species) factors

A major limiting factor in the development of new treatments is the lack of predictive pre-clinical models that accurately reflect human disease with regard to liver histology, pathophysiology, and metabolic abnormalities

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These changes in GSH levels have the potential to yield clinical benefits, as illustrated in Figure 3

Mol Cell Endocrinol 248 , 182191 (2006)