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CRY1/2 Selectively Repress PPAR and Limit Exercise Capacity

The cleavage of AA/AdA-PE-OOHs leads to the accumulation of highly electrophilic secondary oxidation products, such as epoxy, oxo- or aldehyde groups, which could cause the formation of pores in the lipid bilayer, and rupture of the plasma membrane ( In brief, lipid peroxidation forms toxic lipid free radicals and serves as a trigger for ferroptosis, especially when excess iron ions exist in the cytoplasm

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Genetic data from India and Pakistan were obtained from open-access whole-genome sequencing data [5]