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In the case of eNOS uncoupling due to relative intracellular BH 4 deficiency, this phenomenon could be prevented by simultaneous overexpression of the BH 4 synthesizing enzyme GTP cyclohydrolase I, which has been demonstrated in isolated cells 92 and in atherosclerotic animal models

Nature 327 239242

Phase 2 trials are planned for 2006

Therapeutic potential of natural compounds acting through epigenetic mechanisms in cardiovascular diseases: current findings and future directions

Stacking of doxorubicin on folic acid-targeted multiwalled carbon nanotubes for in vivo chemotherapy of tumors

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