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Although there are compelling data in favor of Hcy as a modifiable risk factor, the debate regarding the significance of Hcy mediated health effects is still ongoing

273 The discrepancy between these studies is due to varied autophagy-inducing systems and levels of ROS 274 (Table 1, Fig

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Window-of-opportunity and evolution-informed trials provide additional resolution by capturing early pharmacodynamic effects, clonal selection, and immune remodeling within intact tumor ecosystems (185, 217, 218)