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This resistance is mediated by different causes and mechanisms, (that are resumed in Table 1 and excellently summarized in different review articles [78].One of the most obvious and intriguing mechanisms of resistance involves alteration in CD38 expression, a crucial factor in both primary and acquired resistance to anti-CD38 therapies

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ORF33 and ORF38 of Kaposis sarcoma-associated herpesvirus interact and are required for optimal production of infectious progeny viruses

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6.2 Clinical research H 2 has demonstrated antioxidant potential in preclinical studies, and several clinical investigations have explored its application in PD