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doi: 10.1080/00103629809370125 53 YeX

Mitochondrial dysfunction and oxidative stress: a vicious cycle In ME/CFS, elevated ROS and RNS levels are associated with mitochondrial dysfunction by damaging mtDNA and membranes, disrupting the ETC, and triggering mitophagy

the Ras/MEK/ERK pathway, and the PI3K/AKT/mTOR pathways both of which are being investigated as potential therapeutic targets (120)

Interestingly, the expressions of hsa-mir-122-5p, IL6, FGL1, ACADM, LEP, INS, CASP1, NLRP3, ACAT1, TNF, ELAVL1, IGFBP7, ADAM9, GDF15, ACADVL, ECH1, octanoylcarnitine (octanoylcarnit.), isoleucine, leucine, L-valine, 7-keto-8-aminopelargonic acid (7-keto-8-amino.), creatinine, decanoylcarnitine (decanoylcarnit.), and hexanoylcarnitine (hexanoylcarnit.) were found to be upregulated in DCM, while those of hsa-mir-30c-5p, hsa-mir-30d-5p, hsa-mir-22-3p, GSTM2, GPX3, GSTM3, ADIPOQ, HADH, PRDX2, PRDX1, SERPINA1, A2M, PRDX6, APOA1, APCS, NPPA, bilirubin, butyric acid (butyrate), alanine, glutamine, and cytidine triphosphate (ara-CTP) were found to be downregulated in DCM

By donating electrons to free radicals, glutathione renders them less reactive and less likely to cause damage to our cells

The main differences across comparison categories are: Mechanism of Action: advantage goes to Dihexa