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Therefore, targeting the metabolic vulnerabilities of KRAS-driven tumors, such as glycolysis, glutamine metabolism, and lipid synthesis, has been considered as a novel therapeutic strategy [3]

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Processes, 11 (5), 1299

Mol Biol Rep 42:947962 Aller I, Rouhier N, Meyer AJ (2013) Development of roGFP2-derived redox probes for measurement of the glutathione redox potential in the cytosol of severely glutathione-deficient rml1 seedlings